New PDF release: Bacterial Adhesion to Host Tissues: Mechanisms and

By Michael Wilson

ISBN-10: 0511063318

ISBN-13: 9780511063312

ISBN-10: 0521801079

ISBN-13: 9780521801072

Adhesion is the the most important first level in any infectious illness. it's for that reason very important to completely comprehend the mechanisms underlying bacterial adhesion in order that we are able to increase tools of protecting our basic (protective) microflora, and of stopping pathogenic micro organism from beginning an infectious method. This publication describes the bacterial buildings accountable for adhesion and the molecular mechanisms underlying the adhesion method. a special characteristic is that it additionally bargains with the implications of adhesion for either the adherent bacterium and the host cell/tissue to which it has adhered.

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Additional resources for Bacterial Adhesion to Host Tissues: Mechanisms and Consequences (Advances in Molecular and Cellular Microbiology)

Example text

J. and Höök, M. (2000). Molecular basis of adherence of Staphylococcus aureus to biomaterials. In Infections Associated with Indwelling Medical Devices, 3rd edn, ed. A. L. Bisno, pp. 27–39. Washington, DC: American Society for Microbiology Press. J. and Höök, M. (2000). Cellular invasion by Staphylococcus aureus involves a fibronectin bridge between the bacterial fibronectin-binding MSCRAMMs and host cell ␣1 integrins. European Journal of Cell Biology 79, 672–679. P. E. (2000). Identification of plasma proteins adsorbed on hemodialysis tubing that promote Staphylococus aureus adhesion.

Lindberg, M. and Höök, M. (1996). A monoclonal antibody enhances ligand binding of fibronectin MSCRAMM (adhesin) from Streptococcus dysgalactiae. Journal of Biological Chemistry 271, 1371–1378. J. F. (1997). Crystal structures of fragment D from human fibrinogen and its crosslinked counterpart from fibrin. Nature 389, 455–462. , Reilly, D. J. (1996). Solution structure of the E-domain of staphylococcal protein A. Biochemistry 35, 1558–1569. , Zahradka, C. and McGavin, M. (1997). Identification of D motif epitopes in Staphylococcus aureus fibronectin-binding protein for the production of antibody inhibitors of fibronectin binding.

1) as well as via a selectin-like function of pertussis toxin. Up-regulation of receptors has also been observed during malaria infection. Brain endothelium from patients dying of malaria expressed several of the implicated malarial receptors (CD36, intercellular adhesion molecule (ICAM) 1, endothelial leucocyte adhesion molecule (ELAM) 1, vascular cell adhesion molecule (VCAM) 1) not observed in uninfected brain tissue and may result from increased levels of tumour necrosis factor (TNF) ␣ found in malaria patients (for a review, see Virji, 1996a).

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Bacterial Adhesion to Host Tissues: Mechanisms and Consequences (Advances in Molecular and Cellular Microbiology) by Michael Wilson


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